Health1UP2 · Differentiated health impacts of primary and secondary ultrafine particles
Horizon 2020 — Marie Skłodowska-Curie Actions
- Duration
- 2017-09-07 → 2019-09-06
- EU contribution
- €183,455
- Participants
- 1
- Scheme
- MSCA-IF-EF-ST
Lines connect the coordinator with its partners.
Results in brief
Differentiated health impacts of primary and secondary ultrafine particles
During the past few decades, population studies have consistently linked the exposure to particulate matter (PM) in ambient air to various negative health effects. Traditionally, these studies have focused in exposures to mass concentrations of PM with a diameter ≤10 and ≤2.5 µm (PM10 and PM2.5, respectively) because they are extensively measured across Europe. However, negative health effects are expected to be enhanced with decreasing particle size, since the upper airways are inefficient at filtering ultrafine particles (UFP, PM ≤ 0.1 µm). Recent research indicates sufficient reason to believe that UFP are responsible for negative health effects otherwise attributed to larger size fractions of PM. The answer to this question is important as it would lead to authorities having to control different sources of particles. Unlike PM10 or PM2.5, UFP contribute very little to PM mass, but they are dominant at particle number concentrations (PNC). UFP might have different origins, which is also associated with their size.Source apportionment techniques, such as Positive Matrix Factorization (PMF) have been extensively applied to identify and quantify the sources contributing to PM mass concentrations. No previous study has tried to tackle the fundamental issue of identifying associations between the different sources of UFP, rather than using surrogates for primary combustion emissions such as black carbon (BC) or proximity to mayor highways. Moreover, although a growing literature is pointing towards UFP as the main culprit of human health impairment, there are currently no legal ambient standards for UFP. Future revisions of the Air Policy by the European Commission should include discussions about the inclusion of new standards for other metrics and pollutants, such as UFP and Black Carbon (a good traffic tracer). Therefore, further information on the health effects of UFP is highly needed. Hence, this study aims to identify the different sources contributing to ultrafine PNC and to disentangle the health effect associated with each of these sources to get a full picture of the dimension of the threat that UFP is posing to Public Health. The main objectives of the Health1UP2 (Differentiated health impacts of primary and secondary ultrafine particles) are: - To identify and quantify the sources of UFP in the cities of London (UK), Barcelona (Spain), Zurich (Switzerland) and Helsinki (Finland) based on long time-series of size-segregated UFP (particle number size distributions) and other pollutants. - To determine the health impacts (with mortality and hospital admissions as outcomes) of the exposure to the different sources of UFP in London, Barcelona, Zurich and Helsinki. The results of this work will (i) serve as indication to the corresponding authorities of which air pollutants should be included in future EU legislation about air quality (ii) add insight to the growing body of literature on the health effects of UFP (iii) tackle an important issue not being considered yet: the health effects of UFP according to their origin.
Data: CORDIS, © European Union
Project objective
Exposure to particulate matter (PM) has been linked to negative health effects. Traditionally attributed to PM<10µm and<2.5µm, negative health effects may be enhanced with decreasing particle size, suggesting that ultrafine particles (UFP, PM<0.1µm) might be responsible for what has previously been attributed to larger size fractions of PM.According to their origin, UFP can be primary (directly emitted to the atmosphere) or secondary (formed in the atmosphere from precursor gaseous pollutants). In central European cities, UFP particle concentrations (N) are mainly affected by road traffic emissions, as evidenced by the parallel variation of N and Black Carbon (BC, traffic tracer) concentrations. However, in European regions with high insolation, the maximum N is observed at midday (when BC is low) due to photochemical nucleation. A methodology developed by Rodriguez et al. (2007) allows the quantification of the primary (N1) and the secondary (N2) contribution to total N. Depending on origin and size, UFP may differently affect human health but no study has yet tackled this key issue. Moreover, there are currently no legal ambient standards for UFP. The main objectives of Health1UP2 (Differentiated health impacts of primary and secondary ultrafine particles) are (i) to uncover the differences in N1 and N2 in the cities of London (UK), Barcelona (Spain), Zurich (Switzerland) and Helsinki (Finland), based on long time-series of size-segregated UFP and BC data, and (ii) to determine the health impacts (mortality and hospital admissions as outcomes) of the exposure to differentiated N1 and N2 in different size-ranges in these cities.This research requires an international collaboration, since different meteorological conditions, pollutant emissions and other parameters influence divergently on N1 and N2. The results of this work will serve as indication to the corresponding authorities of which pollutants should be included in future EU legislation about air quality.
Original text from CORDIS.
Participants
- KING'S COLLEGE LONDON · LondonCoordinatorUnited Kingdom
Links
- View on CORDIS
- DOI: 10.3030/747882
- http://www.erg.kcl.ac.uk/Research/home/projects/Health-up-differential-health-impacts.html
Data: CORDIS, © European Union
